Basic Medical Sciences for MRCP Part 1 by Philippa J. Easterbrook MB BChir BSc(Hons) FRCP DTM&H

By Philippa J. Easterbrook MB BChir BSc(Hons) FRCP DTM&H MPH

Written for applicants sitting their MRCP half 1 exam, this revision specializes in the routine issues which arise within the questions. The e-book additionally encompasses a bankruptcy on medical pharmacology (which on my own debts for as much as 30% of the questions), taking a look at facets of drug-induced disorder and drug interactions. eventually there's a bankruptcy on statistics and epidemiology which is never coated in different texts, yet is usually incorporated within the exam.

  • Helps MRCP half 1 applicants organize for and move their exam.
  • Addresses an more and more vital subject within the exam.
  • Addresses an issue that's important to passing the examination, yet which so much applicants are poorly ready for.
  • Covers all of the suitable uncomplicated technological know-how matters plus contains scientific pharmacology.
  • Is of use to applicants learning for different postgraduate checks resembling PLAB, USMLE and MRCPCH.
  • Is the 1st publication of its sort within the club industry and is now considered as crucial for examination preparation.

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Alzheimer's and Parkinson 's disease • HIV disease (CD4+ cells die through programmed cell death). Insufficient • Cancer • Autoimmunity. 35 Cell senescence or ageing • May be associated with repetitive DNA sequences, called telomeres, that are found at the end of chromosomes. They are not replicated in the same way as chromosomal DNA, but are synthesized by the enzyme telomerase. • Cell ageing is associated with shortening of telomeres, possibly due to the absence of telomeres in somatic cells.

Familial hypercholesterolaemia One of the few inborn errors of metabolism inherited as a dominant trait. Due to mutations which d isrupt the synthesis, function or recycling of the low density lipoprotein receptor, there is poor uptake of LDL-bound cholesterol and high serum levels. Martan's syndrome Results from mutations in a gene called fibril/in, the protein product of which is the major component of extracellular microfibrils and is widely distributed in connective tissue throughout the body.

Psittaci C. trachomatis C. } 0_J 0 en 0 a: Characteristic Exotoxin Produced by: Certain Gram +ve and -ve bacteria Gram - ve bacteria Composed of: Secreted polypeptide Stability Endotoxin Heat labile Lipid A in lipopolysaccharide in cell wall Heat stable Antigenicity Highly antigenic. Toxoids Poorly antigenic Biological actions Inhibits protein synthesis LPS molecule induces TNFa Clinical effects Various i Production of cAMP Anthrax toxin Cholera toxin: secretory diarrhoea E. coli enterotoxin: secretory diarrhoea Pertussis toxin : fluid and electrolyte loss, mucus secretion in respiratory tract Inhibition of protein synthesis Diphtheria toxin: cell death P.

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