By Woodruff T.K.
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FIG. 7-2. 7. Otherwise, whole body imaging showed physiological FDG uptake. An earlier thyroid evaluation was available from March 2009 that had been performed in a regional institute in the “Salzkammergut” in Upper Austria. The FDG-avid lesion was noted to be a cold nodule by Technetium-scintiscan, and fine-needle-aspiration cytology had shown oncocytic transformation of thyrocytes. Serum calcitonin screening had been negative in the patient. 3. THE MOST LIKELY DIAGNOSIS IS: a. b. c. d. Cushing´s disease (pituitary origin) Cancer-related Cushing´s syndrome Ectopic ACTH production Erroneous laboratory values no hypercortisolism e.
Of non-cardiogenic pulmonary edema, which can be reproduced in humans by the injection of norepinephrine (6). Capillary hypertension in the lung has been suggested to be a cause of interstitial pulmonary edema, a finding based on experiments in which animals were injected with excessive epinephrine (7). Although to the best of our knowledge, the coexistence of PTS and non-cardiogenic pulmonary edema in a single individual with pheochromocytoma has never been reported, these two manifestations of pheochromocytoma might share some of the same pathogenesis.
USPSTF recommendation level: C; evidence, fair (2|⊕○○○). 2. 1. Because thyroid receptor antibodies (thyroid receptor stimulating, binding, or inhibiting antibodies) freely cross the placenta and can stimulate the fetal thyroid, these antibodies should be measured by 22 wk gestational age in mothers with: 1) current Graves’ disease; or 2) a history of Graves’ disease and treatment with 131I or thyroidectomy before pregnancy; or 3) a previous neonate with Graves’ disease; or 4) previously elevated TRAb.
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